DC-SIGN and DC-SIGNR interact with the glycoprotein of Marburg virus and the S protein of severe acute respiratory syndrome coronavirus.

نویسندگان

  • Andrea Marzi
  • Thomas Gramberg
  • Graham Simmons
  • Peggy Möller
  • Andrew J Rennekamp
  • Mandy Krumbiegel
  • Martina Geier
  • Jutta Eisemann
  • Nadine Turza
  • Bertrand Saunier
  • Alexander Steinkasserer
  • Stephan Becker
  • Paul Bates
  • Heike Hofmann
  • Stefan Pöhlmann
چکیده

The lectins DC-SIGN and DC-SIGNR can augment viral infection; however, the range of pathogens interacting with these attachment factors is incompletely defined. Here we show that DC-SIGN and DC-SIGNR enhance infection mediated by the glycoprotein (GP) of Marburg virus (MARV) and the S protein of severe acute respiratory syndrome coronavirus and might promote viral dissemination. SIGNR1, a murine DC-SIGN homologue, also enhanced infection driven by MARV and Ebola virus GP and could be targeted to assess the role of attachment factors in filovirus infection in vivo.

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عنوان ژورنال:
  • Journal of virology

دوره 78 21  شماره 

صفحات  -

تاریخ انتشار 2004